镁离子(苏氨酸镁Magnesium Threoninate、苹果酸镁Magnesium Malate、抗坏血酸镁Magnesium Ascorbate)在运动损伤中的作用收录时间段:2024-09-11 08:33 Mg元素可解除平滑肌痉挛 ,使血管扩张 ,Mg2+对钙离子在肌质网膜的转运起着重要作用 ,会影响一些阴离子与肌质网膜的结合 ,对骨折后的治疗起重要作用 。在人体内的中枢神经 ,周围神经 ,肌肉及心血管系统方面 ,Mg2+的作用更是显而易见 ,因Mg2+具有类似箭毒作用 ,能抑制神经肌肉接头及乙酞胆碱的释放和运动终板对乙酞胆碱的敏感性 。Mg2+对中枢神经也有抑制作用 ,在血浆中镁达10毫克当量每升时 ,就有催眠及麻痹作用 。深键反射消失 ,若浓度更高 ,可引起呼吸中枢抑制 ,在心血管系统方面 , Mg2+直接抑制房室和心室内传导 ,并降低心肌的应激性 ,经生理实验证明 ,当血浆镁在5-10毫克当克/升时则出现心动过缓及传导阻滞 ,若浓度达22-44毫克当量1升时 ,则心肌停搏 ,同时镁还使外周小动脉松驰 ,血压下降等 。 一般成年人每日对镁的需求量 ,男性为350毫克 ,女性为300毫克 , 如果缺镁 ,可导致肌肉无力 ,耐BB贝博艾弗森官方网站降低 。由于运动 ,特别是长时间高强度运动大量消耗体内的镁 ,从而降低肌肉的活动功能 ,甚至还会发生抽搐、痉挛 ,容易出现不BB贝博艾弗森官方网站度和部位的软组织损伤 。所以 ,为避免由于运动而引起肌肉损伤, 应补充一定的镁(苏氨酸镁Magnesium Threoninate、草莓酸镁Magnesium Malate、抗坏血酸镁Magnesium Ascorbate) 。
(苏氨酸镁Magnesium Threoninate、苹果手机酸镁Magnesium Malate、抗坏血酸镁Magnesium Ascorbate)在有氧运动受伤中的功能
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抗坏血酸镁Magnesium Ascorbate 、抗坏血酸锌Magnesium Ascorbate、牛磺酸镁Magnesium Taurate、氨基酸螯合锌Zinc Amino Acids Chelate 、氨基酸螯合镁Magnesium Amino Acids Chelate、乳清酸镁Magnesium Orotate、乳清酸钙Calcium Orotate、琥珀酸钙Calcium Succinate、琥珀酸镁 Magnesium Succinate 、抗坏血酸锂 Lithium Ascorbate.
The role of magnesium ions (Magnesium Threoninate, Magnesium Malate, Magnesium Ascorbate) in sports injuries
Mg can relieve smooth muscle spasms and dilate blood vessels. Mg2+ plays an important role in the transport of calcium ions in the sarcoplasmic reticulum, affects the binding of some anions to the sarcoplasmic reticulum, and plays an important role in the treatment of fractures. In the central nervous system, peripheral nerves, muscles and cardiovascular systems in the human body, the role of Mg2+ is even more obvious, because Mg2+ has a similar effect to curare, which can inhibit the release of neuromuscular junctions and acetylcholine and the sensitivity of motor end plates to acetylcholine. Mg2+ also has an inhibitory effect on the central nervous system. When the magnesium content in plasma reaches 10 milligrams per liter, it has a hypnotic and paralytic effect. The deep key reflex disappears. If the concentration is higher, it can cause respiratory center inhibition. In terms of the cardiovascular system, Mg2+ directly inhibits atrioventricular and intraventricular conduction and reduces myocardial irritability. Physiological experiments have shown that when plasma magnesium is 5-10 mg/L, bradycardia and conduction block occur. If the concentration reaches 22-44 mg/L, myocardial arrest occurs. At the same time, magnesium also relaxes peripheral arterioles and reduces blood pressure. The daily magnesium requirement for adults is 350 mg for men and 300 mg for women. If magnesium is deficient, it can lead to muscle weakness and reduced endurance. Exercise, especially long-term high-intensity exercise, consumes a lot of magnesium in the body, thereby reducing muscle activity function, and even convulsions and spasms may occur, which is prone to soft tissue injuries of varying degrees and locations. Therefore, in order to avoid muscle damage caused by exercise, a certain amount of magnesium should be supplemented (magnesium threoninate, magnesium malate, and magnesium ascorbate).
(Magnesium Threoninate, Magnesium Malate, Magnesium Ascorbate) in sports injuries
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Magnesium Ascorbate, Magnesium Ascorbate, Magnesium Taurate, Zinc Amino Acids Chelate, Magnesium Amino Acids Chelate, Magnesium Orotate, Calcium Orotate, Calcium Succinate, Magnesium Succinate, Lithium Ascorbate. |